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To understand mechanisms underlying GSH deficiency in older HIV patients and whether GSH deficiency contributes to impaired mitochondrial fuel oxidation in these patients, we proposed and tested the following hypotheses: 1) GSH deficiency results from impaired synthesis due to a deficiency of its precursor amino acids and can be improved by supplementing with GSH precursors and 2) correcting GSH deficiency would improve fasted and fed mitochondrial fuel oxidation, insulin sensitivity, body composition, and muscle strength

ESG may be considered for class I patients who are ineligible for or decline surgery

Identification of the NAD+-binding fold of glyceraldehyde-3-phosphate dehydrogenase as a novel RNA-binding domain

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Oral formulations work for many patients but typically require higher doses to achieve blood levels comparable to injections, and some patients experience gastrointestinal side effects from higher oral volumes