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Laboratory protocols demonstrate that under optimal dosing conditions, the peptide eliminates senescent cells while destroying an undetectable number of healthy cells, representing exceptional selectivity compared to earlier senolytic approaches

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However, Lip-1 concurrently inhibits the enzymatic activity of CYP2D6 (a member of the cytochrome P450 superfamily of drug-metabolizing enzymes), which may render Lip-1 unsuitable for clinical trials beyond its role in suppressing ferroptosis [54]

However, unlike GHK-Cu it doesnt stimulate collagen production